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Their More moderen work even more uncovered that the atypical GTPase domain of Fbx4 could serve as a substrate-binding motif for that SCF E3 ligase advanced and bind to a globular area of TRF1 for its ubiquitination and degradation. These results provide clues on the underlying system for controlling TRF1, https://arthurzjyob.dreamyblogs.com/28655863/a-secret-weapon-for-gençlik-aşısı-nedir

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